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I wrote a non-zero amount, but also not as much as I would have liked

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However, at pharmacological concentrations achieved with therapeutic GLP-1 receptor agonists (100 pM10 nM), -arrestin-2 becomes essential for sustained signaling, specifically mediating extracellular signal-regulated kinase (ERK) activation and CREB phosphorylation that promotes pancreatic -cell survival and insulin synthesis