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While all GLP-1 agonists share some common gastrointestinal effects, the triple agonism of retatrutide introduces glucagon receptor-mediated effects that the others do not produce

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found that non-diabetic human islets consist of three distinct populations of alpha-cells that differ based on proglucagon-derived peptides: one population that produces GLP-1, one that produces glucagon, and one that produces both GLP-1 and glucagon ( Figure 3 )

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