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Regarding potential solutions related to the treatment, panelists reached consensus on the suitability and feasibility of the promotion of a more patient-centered approach to treatment selection (#3 in Table S3), and the implementation of treatment optimization protocols in people with T2D with recent hospital admissions due to CVD (#11)

Research suggests that combining lifestyle changes, medication, and diet changes to improve the levels of GLP-1 could help manage type 2 diabetes more effectively and lower the risk of long-term complications associated with the condition

PI3K activates protein kinase B (Akt), which inactivates several important substrate proteins, such as glycogen synthase kinase 3 (GSK-3)18 and forkhead box O,19 and activates mammalian target of rapamycin (mTOR).20 As a result, it modulates some cellular processes, such as cell survival, proliferation, apoptosis, protein synthesis, inflammation, ER stress, mitochondrial function, and autophagy in neurodegenerative disorders.1820 Akt also promotes B-cell lymphoma 2 and B-cell lymphoma extra-large transcription by activating cyclic adenosine monophosphate (cAMP) response element-binding protein (CREB).21 Thereafter, it regulates learning, memory,22 and neuron survival.23 MAPK regulates various cellular activities including proliferation, differentiation, apoptosis or survival, inflammation, and innate immunity.24 Impairment of insulin signaling is common in diabetes-related NDs

van Ruiten et al., 2022)