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Moreover, upon the stimulation of keratinocyte cell suspensions from these patients with TNF and IFN, there was no GLP-1R expression detected, indicating that the expression of GLP1R in psoriatic plaques was from immune cells expressing these receptors
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Type 1 is an autoimmune condition requiring insulin replacement, and GLP-1 medications do not address the underlying pancreatic beta-cell destruction
BMI, body mass index
doi: 10.1111/dom.13838 25
