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However, based on the discussion of the mechanisms of NADPH oxidase activation above, potential mechanisms of deactivation can include the dephosphorylation of phox subunits, activity of GAPs on Rac proteins, and disassembly of the complex

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[DOI] [PMC free article] [PubMed] [Google Scholar] 66.Sperandio S, Poksay K, De Belle I, et al

In the CNS, the presence of circulating proinflammatory cytokines promotes dysfunction in glutamate reuptake and a decrease in dopamine, both related to the development of fatigue and both capable of inducing an increase in oxidative stress through different pathways 57

By scavenging excessive ROS, polysaccharides can prevent the activation of pro-tumorigenic pathways (e.g., NF-B and MAPK) that are driven by oxidative stress (136)