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The authors identify a cluster of proteins that target human complement and bind plasminogen, revealing a conserved structural fold and a multifunctional immune evasion strategy that enables survival of Borrelia recurrentis in the human host.

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Following stimulation with PMA, human isolated leucocytes, but not mononuclear cells, released enzymatically active PAD, the activity of which was abolished upon pre-incubation of the cells with the glutathione reductase inhibitor 2-AAPA