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FB1-induced DNA damage and hepatocarcinogenesis in experimental models can be modulated by a variety of factors including nutrients, chemopreventive agents, and other factors such as food restriction and viral infection, as well as genetic polymorphismsn rat C6 glioma cells, FB1 inhibits protein synthesis, causes DNA fragmentation and cell death, increases 8-hydroxy-2-deoxyguanosine, induces LPO, and cell cycle arrest [119]

For example, it increases the number of B cells [118] and decreases chronically elevated pro-inflammatory cytokines, such as IL-6, through estrogen supplementation [119]

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In this study, it was found that genes encoding TGA and PR1 were upregulated after plant treatment, promoting salicylic acid signal transduction, which may help to break the inhibition effect of cadmium stress on potato growth