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Many demyelinated axons survive during a relapse, and these can become chronically demyelinated axons, in which case axonal mitochondria develop compensatory mechanisms to cope with the lack of myelin
doi: 10.33698/NRF0159 22
Mitochondrial uncoupling protein 2 is up-regulated in human head and neck, skin, pancreatic, and prostate tumors
Experimental models demonstrate that upregulating EAAC1 function enhances cysteine uptake, elevates GSH levels, and significantly reduces neuronal apoptosis and infarct size following ischemic insult
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