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These findings strongly indicate a protective role for ATP7A-dependent copper release in neurons and may partly account for the neurodegeneration seen in ATP7A-linked disorders such as Menkes disease, which shows decreased function of this protein

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53 Theories to explain why pulmonary GSH could be depleted in some infants include decreased cord cysteine concentrations, seen in earlier gestational age, that may impair GSH synthesis as intracellular GSH depends on the availability of cysteine

Therapeutic strategies based on enhancing ferroptosis are being tested in some cancers, including chemical drugs and nano drugs

As a powerful antioxidant, GSH is particularly important as a regulator of mitochondrial metabolism and a free radical scavenger that limits oxidative damage to cellular components