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Iron metabolism and the role of HFE gene polymorphisms in Wilson disease

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In summary, we identified ER stress as the main adaptive stress response caused by AQ-QI and DEAQ-QI and evaluated the protective role of GSTP1 using a transfected HepG2 cell model

Although clinical translation of these approaches remains challenging, current findings suggest that simultaneously enhancing mitochondrial and peroxisomal antioxidant capacity may provide a more effective strategy to maintain ovarian organelle network homeostasis