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For example, the functioning of the cell actin is regulated by reversible S-glutathionylation, and disturbance in S-glutathionylation changes the structural organization of stress fibrils of the actin cytoskeleton [22]

A recent study also shows that blocking IGF1R-mediated signaling reduces cell proliferation and increases cells sensitivity to the destructive effects of ionizing radiation mediated by down-regulation of BRCA2, a mediator of HR, suggesting that IGF1R is a target for enhancing tumor sensitivity to radiotherapy (Venkatachalam et al., 2017)

Importantly, oxidative stress and impaired methylation are amenable to metabolic interventions

Treatment of Vitamin B12 deficiency

Exploring the Role of neuroplasticity in development, aging, and neurodegeneration