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In other ASD models, such as 15q dup mice [34], Oprm1 -KO mice [42], Oxtr -KO mice [33], and C58/J mice [38], oxytocin selectively improved social deficits, without influencing other ASD symptoms

10.1186/1742-2094-1-10 J

Preclinical research exists, but human studies are still limited

101), it is conceivable that pharmacological up-regulation of PINK1/Parkin-mediated mitophagy could become a potential therapeutic alternative for APAP toxicity

One study found that low homocysteine is associated with idiopathic peripheral neuropathy, a condition that damages nerves.6 This makes sense, as the body needs homocysteine to produce glutathione, which supports the nerves healthy response to oxidative stress