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Artery Dis

The function of TMAO in causing endoplasmic reticulum (ER) stress and insulin resistance in diabetes is well-documented, with TMAO levels greater in type 2 diabetics

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Int J Chronic Dis

The therapeutic arsenal has since expanded to include brolucizumab and faricimab, with the phase 3 TENAYA and LUCERNE trials demonstrating that faricimab with a treat-and-extend protocol delivers strong visual and anatomical outcomes at 2 years, offering sustained disease control with fewer injections (Khanani et al., 2024)
