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From the perspective of metabolism, CAP potentiated high-rate apoptosis of chronic myeloid leukemia cells, primary CD34 + hematopoietic stem and progenitor cells, while sparing their normal counterparts by disrupting the skewed metabolism of tumor cells as a result of ultra redox sensitivity [222]

FIGURE 1 FIGURE 2 Oxidative stress effect on sepsis pathology Oxidative stress plays an essential role in the pathogenesis and progression of sepsis, primarily through the overproduction of ROS and impairment of antioxidant defence mechanisms

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