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treated 99 patients with Tepotinib

146 The authors also discovered a point mutation that affected codons 221250, and disrupted most of the CD38 epitopes bound by daratumumab, thus facilitating the evasion of MM cells from daratumumab

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HGF targeting antibodies, including rilotumumab (AMG-102), ficlatuzumab, TAK-701, and YYB-101, bind to HGF and prevent c-Met attachment, leading to the induction of synergistic anti-tumor responses combined with other TKIs and EGFR inhibitors [162, 172, 178, 179]
