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[88] included seven prospective and retrospective studies in both adult and pediatric patients with non-acetaminophen ALF
Galgano et al., 2017)
In NSCLC, persistently activated NRF2 creates distinct enhancers at gene loci that are not typically regulated by NRF2 under normal physiological conditions
They modulate immune cell polarization, reverse drug resistance by targeting key signaling and metabolic pathways, and regulate cell death processes such as apoptosis, autophagy, and ferroptosis
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