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Given that the D1-like activation of GluN2B and predominant NMDAR contribute to synaptic plasticity, it would be important to investigate whether MPH may inhibit NMDAR plasticity for neurons expressing D1-like receptors

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92 In this mechanism, bacterial GSH is an essential cofactor, and mutants that are defective in GSH biosynthesis cannot inactivate fosfomycin, even though they possess the resistance gene

For this reason we believe the most effective plasmid design should place Gpx closest to the promoter, where it has the highest probability of being fully transcribed by RNA polymerase, and GR furthest from the promoter, since high concentrations of GR appear to be less critical given its efficacy