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Conclusion The objective of this investigation was to employ network pharmacology to pinpoint the potential proteins which may be involved in the oxidative stress pathways in response to either epilepsy or epilepsy treatment using two antiseizure medicationscarbamazepine and valproic acid

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It works internally via gene activation, not surface exfoliation

However, UIBC did not exhibit significant changes (Fig

Synaptic GABA A Rs are thought to modulate rapid phasic inhibition while extrasynaptic receptors are typically thought to influence tonic inhibition, which is important for proper regulation of the excitatory-inhibitory balance