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Furthermore, ER stress is acknowledged as a secondary consequence of metabolic dysfunction, frequently caused by the excessive buildup of reactive oxygen species (ROS) and disruptions in intracellular calcium levels

BAY-1082439, a chemical molecule undergoing phase I clinical trials in patients with advanced cancer, can efficiently inhibit the expression of P-gp and BCRP by selectively inhibiting PI3K 110 and 110, and slightly suppress the efflux effect of P-gp and BCRP (118)

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Pande and Flora [69] reported that a combination of lipoic acid and thiol chelator was able to alleviate lead toxicity, and in particular, it reduced oxidative stress and lead levels in the brain