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In rodents, BPC-157 accelerates healing in alkali-burn wound models, coinciding with increased ERK1/2 activity in regenerating tissues, suggesting that this signaling axis contributes not only to cellular dynamics in vitro but also to tissue-level repair responses in vivo [26, 55]

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In Table 1 the threshold laboratory parameters and the probability of Wilson disease according to the clinical and laboratory findings is summarized

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