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it modifies ovarian function and increases the risk of developing metabolic and cardiovascular diseases (Abraham Gnanadass et al., 2021
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Figure 6 Discussion Non-genetic mechanisms have recently emerged as important drivers of cancer therapy failure, whereby some cancer cells enter a reversible drug-tolerant persister state in response to treatment ( et al
L-2-oxothiazolidine-4-carboxylic acid (OTC) treatment attenuated plasma and tissue P-selectin levels, suggesting that the upregulation of P-selectin in hyperhomocysteinemic mice is also regulated by redox mechanisms [61]
