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Its primary structural feature is the actin-binding domain, which allows TB-500 to regulate actin polymerisation the cellular process underpinning cell migration, proliferation, and wound closure

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In endothelial cell models, BPC-157 significantly enhances ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formationprocesses central to tissue regeneration and angiogenesis [26, 55]

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