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[DOI] [PubMed] [Google Scholar] 457.Duman N., Ersoy Evans S., Atakan N

In summary, this study revealed that dysfunction of cellular transport of cytoplasmic FA to mitochondria plays a critical role in UA-induced cardiomyocyte injury and thereby promotes LVR development and identified CPT1B as the key factor influenced by UA

Detection of suppressed maturation of the human COQ5 protein in the mitochondria following mitochondrial uncoupling by an antibody recognizing both precursor and mature forms of COQ5

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Intriguingly, the development of membrane irregularities coincides with growth arrest (Fig 5A and Movie EV1)