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We predict that breast cancer cells treated with ATZ alone should have a greater proliferation rate associated with stronger EGF signaling activity, while the co-treatment of ATZ+antagonist and ATZ+Curcumin should produce limited proliferation and diminished EGF signaling activity

Considering that PSC activation is a key factor in pancreatic fibrosis, we speculated that excess ROS production induced by high glucose may induce PSC activation in vivo

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