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Importantly, overexpression of keap1a or knockdown of hmox1a partially rescued the blood clot formation, suggesting that the disrupted Keap1-Nrf2 signaling is responsible for the venous thrombosis-like phenotypes in smarca5 mutants
Increasing evidence suggests that there are different ASD endophenotypes
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This follows our last paper in NPJ Microgravity, demonstrating enrichment of GPX1 in ExerVs collected from humans after a combined endurance and strength training program
