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It has been shown that Nrf2 modulates the GSH redox state via glutathione reductase regulation
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doi: 10.1016/j.scitotenv.2019.134664 55 StokerTELawsSCCroftonKMHedgeJMFerrellJMCooperRL
This system showed excellent tumor-targeting potential with minimal side effects, offering a promising approach for treating NQO1-overexpressing tumors
In support of this observation, over expression of GS in a yeast model failed to increase glutathione (GSH) levels whereas over expression of GCL did [18]
