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Conclusions These findings identify disrupted GSH homeostasis and inflammatory activation as metabolic features associated with symptomatic TTR-V30M amyloidosis in vivo, supporting a link between redox imbalance and disease manifestation

The sulfur atom of glutathione is close to the OH of Tyr9 in the active site ( a value from 6.7 to 7.2 when Tyr9 was mutated into Phe (Pettersson and Mannervik, 2001)

Accumulated microcracks increase bone brittleness, predisposing patients to atypical femoral fractures in the subtrochanteric or diaphyseal regions following minimal or no trauma

These molecular effects translated into substantial reductions in CRC cell migration and invasion (44)

Metallomics and other omics approaches in antiparasitic metal-based drug research