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In another study on human primary gastric TP53 / PDOs, knocking down the frequently mutated AT-rich interaction domain 1A ( ARID1A ) gene, led to a phenotype resembling ARID1A -mutant gastric cancers, particularly the MSI- (microsatellite instability) and (Epstein-Barr virus) EBV-associated subtypes, where ARID1A mutations are more common (Lo et al., 2021)

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Specifically, TPG-1 potentiated the production of inflammatory cytokines, including nitric oxide (NO), tumor necrosis factor (TNF-), and interleukin-6 (IL-6), in macrophages by activating toll-like receptor 4/nuclear factor kappa B/mitogen-activated protein kinase (TLR4/NF-B/MAPK) signaling in macrophages [118]

Membrane-bound class III peroxidases: unexpected enzymes with exciting functions