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In TNBC cells, acetaldehyde, a major alcohol metabolite, induced cell migration and invasion and increased the phosphorylation of p38, JNK, and NF-B in a manner similar to ethanol [72]

14,15,16,20 NAC is a cysteine prodrug and glutathione precursor, which enters glial cells in exchange for glutamate and is converted to intracellular glutathione, a potent antioxidant and neuroprotective substrate

AE is caused by mutations of the SLC39A4 gene on the chromosome band 8q24 mediates alcohol-induced down-regulation of intestinal tight junction proteins

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