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Description
doi: 10.3390/biom5010194

EOFAD is characterized by early onset, family history, a variety of neurological symptoms, and a more aggressive course compared to sporadic AD (Wu et al

ANE can delay cell senescence by inhibiting PARP1 activity, thereby encouraging SIRT1 activity and increasing NAD + release

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LDHA deficiency is characterized by the complete absence of the M isoform [34, 35]
