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Therefore, any use outside research contexts carries uncertainty regarding both efficacy and safety

It is noteworthy that gene-nutrient interactions, especially between the C677T mutation of the MTHFR gene and folate status and perhaps between heterozygosity for CBS defect and folate status (Lentz et al, 2000), could be involved in the onset of hyperhomocysteinemia

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The geographic distribution of this condition is consistent with positive selection for G6PD deficiency in areas where malaria is common