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Development of a Buffer-Free Enzymatic Method for Hydrolysis of Methyl Esters

Molecular mechanism of epidermal barrier dysfunction as primary abnormalities

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Depletion of Aurora-A co-activators from asynchronous U251 cells, particularly that of BORA, CEP192 and TPX2, led to increased levels of mitotic phosphorylations (PPP1CA-pT320, PRC1-pT481 and Histone-H3-pS10), indicative of delayed mitotic progression (Fig
doi: 10.1016/j.resp.2008.10.008
