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Further, DISC1 is not found to be associated with SZ risk in more recent GWAS studies and its relevance to SZ is therefore questionable [131, 139] Other genetic models are conditional knock-outs for neuregulin and its receptor ErbB4, showing distinct SZ-like characteristics

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Tatekoshi Y, Tanno M, Kouzu H, Abe K, Miki T, Kuno A, Yano T, Ishikawa S, Ohwada W, Sato T, Niinuma T, Suzuki H, Miura T (2018) Translational regulation by miR-301b upregulates AMP deaminase in diabetic hearts

Potential medical applications of the phenomenon of lysinearginine antagonism are listed in Section 7