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They also activate the platelets interacting with neutrophils and induce neutrophil extracellular trap (NET) formation [13]
doi: 10.1016/j.antiviral.2018.07.001 [DOI] [PMC free article] [PubMed] [Google Scholar] 15.Takashita E, Daniels RS, Fujisaki S, et al
Collectively, these findings indicate that the inflammationfibrosis axis represents a central driver in the pathogenesis of dyslipidemia-induced ED and a promising therapeutic target (70, 71)
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L-cysteine metabolism and its nutritional implications
