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Non-radical ROS in disease pathogenesis Mechanisms of pathogenic signaling The roles of non-radical ROS in disease pathogenesis are defined not only by their indiscriminate oxidative power, but more importantly by the precise mimicry or supplementation of their natural, low-flux physiological signaling pathways to orchestrate divergent cellular responses, yielding therapeutic benefit in some contexts while driving pathology in others (Fig

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Physiological analysis showed that under drought stress, FA increased the relative water and chlorophyll contents of oat leaves, enhanced the activity of antioxidant enzymes (SOD, POD, PAL, CAT and 4CL), inhibited the accumulation of malondialdehyde (MDA), hydrogen peroxide (H 2 O 2 ) and dehydroascorbic acid (DHA), reduced the degree of oxidative damage in oat leaves, improved the drought resistance of oats, and promoted the growth of oat plants

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