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Substantiating this mechanism, murine studies with macrophage-specific deletion of myeloid differentiation primary response 88 (MyD88) demonstrated that inhibition of TLR/NF-B-mediated macrophage inflammation significantly attenuates hepatic lipid accumulation and injury in NAFLD models [87]

Samples were collected and stored as described above

ALA can also enhance the developmental potential of oocytes in the mouse by reducing the level of ROS [96]