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Melatonin administration reversed this phenotype by adopting a compensatory mechanism aimed at increasing Bcl-2 protein expression, blocking Bax proapoptotic activity via the SIRT1/NF-kB axis with a consequent and significant inhibition of Cyt C release and the lack of apoptosome formation and caspase 3 activation 37 (Fig
Pdx-1: a promising therapeutic target to reverse diabetes
10.1183/13993003.01126-2025 Eur Respir J
Int J Mol Sci 18(12):2698
[Abstract] Antiviral Res
