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In support of the NRF2 pathway being a potential target for the treatment of ALS, studies have shown that NRF2 overexpression in astrocytes provided neuroprotection against mutated SOD1-induced toxicity and increased the median survival of mutant SOD1 transgenic mice (Vargas et al., 2008

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JFH1 wt replication in TLR3 expressing cells was associated with a substantial induction of IFIT1 mRNA expression, indicative of a strong innate immune response counteracting replication, in line with previously published reports 30,31,32,33 (Fig

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