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Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and Disease

Our analyses displayed that endometriosis could be polyepigenetic and with alterations in specific genes implicated in major signaling pathways contributing to the disease etiopathology (cell proliferation, differentiation, and division [PI3K-Akt and Wnt-signaling pathway], cell division [MAPK pathway], cell adhesion, cell communication, developmental processes, response to hormone, apoptosis, immunity, neurogenesis, and cancer)

Although inflammatory genes such as TNF, IL-6, and IFN did not show drastic changes compared to control, E-cadherin (ECAD) expression significantly decreased within the CS-exposed ALI + Str samples (Supplementary Fig

The results of the Kuybulu research group indicate that elevated levels of malondialdehyde (MDA) and total oxidant (TOS), along with reduced levels of total antioxidant (TAS) and superoxide dismutase (SOD) in kidney tissue, indicate a situation of oxidative imbalance