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Researchers may not be able to conclude that excessive cell death induced by ferroptosis is associated with pathogenic processes based only on the preventive effect of ferrostatin-1 or liproxstatin-1 on cell death 21 , as these compounds are essentially antioxidants and may inhibit other ROS-dependent forms of cell death, such as necroptosis 46

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Notably, our results show that excessive antioxidant thiols originate from the host microbiota

NADPH oxidase-1 plays a crucial role in hyperoxia-induced acute lung injury in mice

For example, combining Semax (peptide) with racetams (small molecule nootropics) can enhance neuroplasticity through both BDNF upregulation and AMPA receptor modulation