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Description
The loss of adrenergic receptor mediated release potentiation in a mouse model of fragile X syndrome
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These processes are believed to play a role in enhancing certain cognitive functions and long term memory capacity
This implies sodium channel inhibition, a blockade of voltage-gated sodium channels (VGSCs) leading to a reversible block of action potential propagation [278], and reduced permeability of cell membranes, thereby blocking depolarization and preventing the conduction of the electrical impulse through which pain occurs [279]
D 66, 125132 (2010)
