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These results highlight that Paxalisib induced organ-specific perturbations and stress responses in the NOCS platform

Mtor Ser2035Cys (105KRP) eliminated binding of rapamycin-FKBP12 to MTOR in contrast with the starting cell line (Figure 2D), while binding of bi-steric inhibitors was not affected

The sample undergoes 4-5 rounds of resuspension and centrifugation to wash away and remove as much unbound non-target material as possible