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Additionally, the lack of methylation impairs the brain's ability to clear out neurotoxic metabolites, allowing them to accumulate and disrupt neural signaling

Considering that this latter neuronal population is involved in sleep-cycle regulation, it is tempting to speculate whether the inhibitory action of OX-A on REM sleep could be mediated, at least in part, by decreasing GHRH mRNA expression in the PVH

These effects appear to arise indirectly through vascular support, inflammation control, and cellular protection rather than direct neurotransmitter stimulation

Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase

CoA-LOT: BN20251112/CA