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Abstract Diabetic cardiomyopathy (DCM) is a common and fatal cardiac complication caused by diabetes, with its pathogenesis involving various forms of cell death and mitochondrial dysfunction, particularly ferroptosis and mitochondrial injury

257,259,260 Multiple mechanisms may act in concert to tip the balance in favor of tau hyperphosphorylation, such as oxidative stress, calcium dyshomeostasis, age-related alterations, or even A-mediated modulation of cell surface receptors at synapses 262

Mice deficient in glutathione transferase zeta/maleylacetoacetate isomerase exhibit a range of pathological changes and elevated expression of alpha, mu and pi class glutathione transferases

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Headache and fatigue can occur, especially during dose increases