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Description
In 1994, Amaras group isolated the human homologs of all three of these transporters and were called EAAT1-3 for Excitatory Amino Acid Transporter [22]
In clinical practice, one of the predominant elements for the choice of treatment options is the patient preference [26,42]
The mean follow-up was over 20 months in both groups, but there was a significant difference between groups favoring patients treated with CS (PRP, 21.18.7 vs
Cell Rep 43(8):114637
The GliSODin group had significantly lower cellular DNA damage as evidenced by a test called Comet Assay. Also, these findings coincided with reduced blood isoprostane levels, another marker of oxidative stress
