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doi: 10.1111/j.1365-2249.2009.03918.x
In conclusion, our structural and functional analyses provide a large body of novel information for defining the molecular mechanism underlying the activation of c-MET in response to HGF and NK1 and paves the way for the eventual therapeutic intervention of diseases caused by aberrant activation of c-MET
SVCT2 overexpression and ascorbic acid uptake increase cortical neuron differentiation, which is dependent on vitamin C recycling between neurons and astrocytes
Comparison of the outcomes of patients with localized esophageal carcinoma receiving bimodal versus trimodal therapy
doi: 10.4103/ijo.IJO_1627_21
