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Several studies have identified genes involved in atherosclerosis (AS), including those encoding proinflammatory cytokines or LDL receptor-related proteins, which are targets for anti-atherosclerosis (anti-AS) gene therapy [15]

Moreover, several GSH-responsive systems have progressed toward early-phase clinical evaluation Fig

On the other hand, a recent study found that miR-206, which targets CXCL11 as a tumor suppressor in PCa, negatively affected PCa cell growth and motility and delayed the cell cycle (Fig

alternatively, can be used as potent inhibitors of APF and the production of AFB1 and other mycotoxins (529)

doi: 10.26355/EURREV_202103_25442 29 NasiA.McArdleS.GaudernackG.WestmanG.MeliefC.RockbergJ.et al