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doi: 10.1158/1535-7163.MCT-19-1020 71 LiX.WangL.YanS.YangF.XiangL.ZhuJ.et al
doi: 10.3389/fcell.2023.1232241 56 ManzDHBlanchetteNLPaulBTTortiFMTortiSV
In endothelial cell models, BPC-157 significantly enhances ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formationprocesses central to tissue regeneration and angiogenesis [26, 55]
When the ventricle fails to eject fully, residual volume remains, and the next diastolic filling adds to it
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