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10.1074/jbc.M005536200 J

2d), they failed to generate any detectable levels of ROS in high DMF concentrations ( f w 40 M) towards the normal cell line (LO2), confirming its excellent cancer selectivity

Current research implicates several interrelated pathogenic mechanisms, including persistent neuroinflammation, mitochondrial dysfunction [27], excessive production of reactive oxygen species (ROS) [28], reduced dopamine synthesis [8], impaired cerebral glucose metabolism [8], and the accumulation of misfolded -synuclein in the form of Lewy bodies [29]

The combination may reduce gut inflammation on multiple levels, including systemically, via cytokine signaling, and through modulation of the intestinal immune response