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Both JNK and ERK mediate HCC cell migration by phosphorylating paxillin at serine residues, which is called an HGF-induced focal adhesion signaling molecule (Kermorgant et al., 2004) to direct fluctuating JNK and paxillin signaling pathways which involve HCC cell migration (Wu et al., 2015) and may be also a specific and safe target for treating HCC patients

Among the central alterations found in fibromyalgia are irregularities in monoaminergic neurotransmission, leading to increased levels of some excitatory neurotransmitters (known as algesic mediators) such as glutamate and substance P [127,128]

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STING activation reprograms tumor vasculatures and synergizes with VEGFR2 blockade

Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice